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VariationsinnativeT1valuesinpatientswithDuchennemusculardystrophywithandwithoutlategadoliniumenhancementIntJCardiovascImagingOriginalPaperEarlyRecent,Sept20,./s--31-z本文由“天纳”临床学术信息人工智能系统自动翻译点击文末“阅读原文”下载本文PDFDuchennemusculardystrophy(DMD)isanX-linkedgeneticdisorderleadingtoprogressiveskeletalandcardiacmyopathy.ElevatedmyocardialT1valuescorrelatewithfibrosisinmostdiseaseprocesses,butDMDskeletalandcardiachistopathologyisdefinedbyfibrofattyreplacementthatmayresultinadecreaseinT1values,duetothelowT1offat.ThestudygoalwastoassessmyocardialT1valuesinDMDpatientswithandwithoutlategadoliniumenhancement(LGE).AretrospectiveanalysiswasperformedonallpatientswithDMDreferredforCMRatourinstitutionfrom7/5/to10/24/.T1measurementswereperformedusingbreath-heldmodifiedLookLockerinversionrecovery(MOLLI)sequencesatthebasalandmid-ventricularlevels.ThecohortwasseparatedintopatientswithoutthepresenceofLGE(LGE?)andpatientswithcurrentorpreviousLGE(LGE+).AtotalofCMRstudieswereanalyzed.TheLGE?group
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